Dermatitis herpetiforme

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dc.contributor.authorArmand Rodríguez, Beatrizspa
dc.contributor.authorÁvila Camacho, Mabel Yanethspa
dc.contributor.authorPierard, Gérald Espa
dc.contributor.authorArrese, Jorge Espa
dc.date.accessioned2020-10-27T14:22:25Z
dc.date.available2020-10-27T14:22:25Z
dc.date.issued2002-08-09
dc.description.abstractLa dermatitis herpetiforme es una enfermedad autoinmune ampollosa subepidérmica caracterizada por una erupción pápulo-vesiculosa pruriginosa asociada a depósitos granulares de IgA a nivel de la dermis papilar detectados por IFD. Esta enfermedad en piel se encuentra asociada a una enteropatía sensible al gluten la cual es generalmente asintomática. El examen microscópico revela microabscesosricos en neutrófilos y eosinófilos a nivel de las papilas dérmicas así como un infiltrado dérmico compuesto de linfocitos T, neutrófilos y eosinófilos. La patogénesis de la enfermedad aún es desconocida y se ha considerado que se trata de una enfermedad mediada por complejos inmunes tipo IgA. El tratamiento con dapsona es efectivo para la enfermedad y asociado a una dieta libre degluten se disminuyen los requerimientos del medicamento. [Armand B, Ávila MY, Pierard GE, ArreseJE. Dermatitis herpetiforme.spa
dc.description.abstractenglishDermatitis herpetiformis is a subepidermal blistering autoimmune disease characterized by a pruritic papulo-vesicular rash associated with granular IgA deposits at the level of the papillary dermis detected by IFD. This skin disease is associated with a gluten-sensitive enteropathy which is generally asymptomatic. Microscopic examination reveals microabscesses rich in neutrophils and eosinophils at the level of the dermal papillae as well as a dermal infiltrate composed of T lymphocytes, neutrophils, and eosinophils. The pathogenesis of the disease is still unknown and it has been considered to be a disease mediated by IgA type immune complexes. Treatment with dapsone is effective for the disease and, associated with a gluten-free diet, the requirements for the medication are reduced. [Armand B, Ávila MY, Pierard GE, ArreseJE. Dermatitis herpetiformis.eng
dc.format.mimetypeapplication/pdfspa
dc.identifier.instnameinstname:Universidad Autónoma de Bucaramanga UNABspa
dc.identifier.issn2382-4603
dc.identifier.issn0123-7047
dc.identifier.repourlrepourl:https://repository.unab.edu.co
dc.identifier.urihttp://hdl.handle.net/20.500.12749/10538
dc.language.isospaspa
dc.publisherUniversidad Autónoma de Bucaramanga UNAB
dc.publisher.facultyFacultad Ciencias de la Saludspa
dc.publisher.programPregrado Medicinaspa
dc.relationhttps://revistas.unab.edu.co/index.php/medunab/article/view/280/263
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dc.relation.referencesBeutner EH, Baughman RD, Austin BM, Plunket RW, BinderWL. A case of dermatitis herpetiformis with IgA endomysialantibodies but negative direct immunofluorescent findings. J AmAcad Dermatol 2000; 43(2 pt 2):329-32spa
dc.relation.referencesDieterich W, Laag E, Bruckner-Tunderman L, Reúnala T, et al.Antibodies to tissue transglutaminase as sepapelogic markersin patients with dermatitis herpetiformis. J Invest Dermatol 1999;113(1):133-6.spa
dc.relation.referencesRose C, Dieterich W, Broker E-V, Schuppan D, Zillikens D.Circulating autoantibodies to tissue transglutaminase differentiatepatients with dermatitis herpetiformis from those with linear IgAdisease. J Am Acad Dermatol 1999; 41(6):957-61.spa
dc.relation.referencesAmerio P, Verdulini R, Grangiacomi M Expression of eotaxina,interleukin 13 and tumour necrosis factor-a in dermatitisherpetiformis. Br J Dermatol 2000; 143:974-8spa
dc.relation.referencesAirula K, Vaalamo M, Reúnala T, Saariglho-Kere UK. Enhancedexpression of interstitial collagenase, stromelysi-1, andurokinase plaminogen activator in lesions of dermatitisherpetiformis. J Invest Dermatol 1995; 105(2):184-9spa
dc.relation.referencesLahteenoja H, Irjala K, Viander M, Vainio E, Toivanen A,Syrjanen S. Oral mucosa is frequently affected in patients withdermatitis herpetiformis. Arch Dermatol 1988; 134:756-8spa
dc.relation.urihttps://revistas.unab.edu.co/index.php/medunab/article/view/280
dc.rights.accessrightsinfo:eu-repo/semantics/openAccessspa
dc.rights.creativecommonsAttribution-NonCommercial-NoDerivatives 4.0 International*
dc.rights.urihttp://creativecommons.org/licenses/by-nc-nd/2.5/co/
dc.sourceMedUNAB; Vol. 5 Núm. 14 (2002): Dermatología; 75-82
dc.subjectCiencias biomédicas
dc.subjectCiencias de la vida
dc.subjectInnovaciones en salud
dc.subjectInvestigaciones
dc.subject.keywordsHealth Scienceseng
dc.subject.keywordsMedicineeng
dc.subject.keywordsMedical Scienceseng
dc.subject.keywordsBiomedical Scienceseng
dc.subject.keywordsLife Scienceseng
dc.subject.keywordsInnovations in healtheng
dc.subject.keywordsResearcheng
dc.subject.keywordsDermatitis herpetiformiseng
dc.subject.keywordsGranular Ig Aeng
dc.subject.keywordsDapsoneeng
dc.subject.keywordsSubepidermal blisterseng
dc.subject.lembMedicinaspa
dc.subject.lembCiencias de la saludspa
dc.subject.lembCiencias médicasspa
dc.subject.proposalDermatitis herpetiformespa
dc.subject.proposalIg A granularspa
dc.subject.proposalDapsonaspa
dc.subject.proposalAmpollas subepidérmicasspa
dc.titleDermatitis herpetiformespa
dc.title.translatedDermatitis herpetiformiseng
dc.type.coarhttp://purl.org/coar/resource_type/c_6501
dc.type.driverinfo:eu-repo/semantics/article
dc.type.hasversionInfo:eu-repo/semantics/publishedVersion
dc.type.hasversioninfo:eu-repo/semantics/acceptedVersion
dc.type.localArtículospa
dc.type.redcolhttp://purl.org/redcol/resource_type/ART

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